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SOD1-G93A/low Mouse Model - QPS Neuropharmacology
SOD1-G93A/low Mouse Model - QPS Neuropharmacology

Tempol improves neuroinflammation and delays motor dysfunction in a mouse  model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text

Early upregulation of cytosolic phospholipase A2α in motor neurons is  induced by misfolded SOD1 in a mouse model of amyotrophic lateral sclerosis  | Journal of Neuroinflammation | Full Text
Early upregulation of cytosolic phospholipase A2α in motor neurons is induced by misfolded SOD1 in a mouse model of amyotrophic lateral sclerosis | Journal of Neuroinflammation | Full Text

Frontiers | Skeletal Muscle-Restricted Expression of Human SOD1 in  Transgenic Mice Causes a Fatal ALS-Like Syndrome
Frontiers | Skeletal Muscle-Restricted Expression of Human SOD1 in Transgenic Mice Causes a Fatal ALS-Like Syndrome

Frontiers | Evaluation of Neuropathological Features in the SOD1-G93A Low  Copy Number Transgenic Mouse Model of Amyotrophic Lateral Sclerosis
Frontiers | Evaluation of Neuropathological Features in the SOD1-G93A Low Copy Number Transgenic Mouse Model of Amyotrophic Lateral Sclerosis

SOD1-positive aggregate accumulation in the CNS predicts slower disease  progression and increased longevity in a mutant SOD1 mouse model of ALS |  Scientific Reports
SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports

IJMS | Free Full-Text | Therapeutic Treatment of Superoxide Dismutase 1  (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone  Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial  Proliferation, and Preserves Monocyte Levels
IJMS | Free Full-Text | Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels

Neuroprotective effects of the cannabigerol quinone derivative VCE-003.2 in  SOD1G93A transgenic mice, an experimental model of amyotrophic lateral  sclerosis - ScienceDirect
Neuroprotective effects of the cannabigerol quinone derivative VCE-003.2 in SOD1G93A transgenic mice, an experimental model of amyotrophic lateral sclerosis - ScienceDirect

Cortical glia in SOD1(G93A) mice are subtly affected by ALS-like pathology  | Scientific Reports
Cortical glia in SOD1(G93A) mice are subtly affected by ALS-like pathology | Scientific Reports

SOD1 (71G8) Mouse mAb | Cell Signaling Technology
SOD1 (71G8) Mouse mAb | Cell Signaling Technology

Decreasing SOD1 in muscle does not improve grip strength in SOD1 G93A... |  Download Scientific Diagram
Decreasing SOD1 in muscle does not improve grip strength in SOD1 G93A... | Download Scientific Diagram

A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores  Function in an ALS Mouse Model - ScienceDirect
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect

ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes  Rapidly Progressive Disease with SOD1-Containing Inclusions: Neuron
ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions: Neuron

Sagittal brain slices from a SOD1 G93A transgenic mouse at terminal... |  Download Scientific Diagram
Sagittal brain slices from a SOD1 G93A transgenic mouse at terminal... | Download Scientific Diagram

Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a  Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis
Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis

Generation and analysis of innovative genomically humanized knockin SOD1,  TARDBP (TDP-43), and FUS mouse models - ScienceDirect
Generation and analysis of innovative genomically humanized knockin SOD1, TARDBP (TDP-43), and FUS mouse models - ScienceDirect

Nutrients | Free Full-Text | Analysis of Feeding Behavior Characteristics  in the Cu/Zn Superoxide Dismutase 1 (SOD1) SOD1G93A Mice Model for  Amyotrophic Lateral Sclerosis (ALS)
Nutrients | Free Full-Text | Analysis of Feeding Behavior Characteristics in the Cu/Zn Superoxide Dismutase 1 (SOD1) SOD1G93A Mice Model for Amyotrophic Lateral Sclerosis (ALS)

Figure 1 from Translational Research on Amyotrophic Lateral Sclerosis  (ALS): The Preclinical SOD1 Mouse Model | Semantic Scholar
Figure 1 from Translational Research on Amyotrophic Lateral Sclerosis (ALS): The Preclinical SOD1 Mouse Model | Semantic Scholar

004435 - SOD1-G93A Strain Details
004435 - SOD1-G93A Strain Details

PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1  mouse. | Semantic Scholar
PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar

Transgenic mice expressing H46R/H48Q/H63G/H120G (Quad) mutant SOD1... |  Download Scientific Diagram
Transgenic mice expressing H46R/H48Q/H63G/H120G (Quad) mutant SOD1... | Download Scientific Diagram

IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A  Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of  Amyotrophic Lateral Sclerosis
IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic Lateral Sclerosis

Preliminary Observation about Alteration of Proteins and Their Potential  Functions in Spinal Cord of SOD1 G93A Transgenic Mice
Preliminary Observation about Alteration of Proteins and Their Potential Functions in Spinal Cord of SOD1 G93A Transgenic Mice

Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic  lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular  atrophy protein to nuclear Cajal bodies. | Semantic Scholar
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar

CuATSM improves motor function and extends survival but is not tolerated at  a high dose in SOD1G93A mice with a C57BL/6 background | Scientific Reports
CuATSM improves motor function and extends survival but is not tolerated at a high dose in SOD1G93A mice with a C57BL/6 background | Scientific Reports

Neuroprotective effects of siRNAp11 in the SOD1-G93A mouse model of... |  Download Scientific Diagram
Neuroprotective effects of siRNAp11 in the SOD1-G93A mouse model of... | Download Scientific Diagram

SOD1-positive aggregate accumulation in the CNS predicts slower disease  progression and increased longevity in a mutant SOD1 mouse model of ALS |  Scientific Reports
SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports

SOD1-G93A/low Mouse Model - QPS Neuropharmacology
SOD1-G93A/low Mouse Model - QPS Neuropharmacology

Knockout of reactive astrocyte activating factors slows disease progression  in an ALS mouse model | Nature Communications
Knockout of reactive astrocyte activating factors slows disease progression in an ALS mouse model | Nature Communications

Reducing SOD1 expression prolongs survival in SOD1 G93A mice. SOD1 G93A...  | Download Scientific Diagram
Reducing SOD1 expression prolongs survival in SOD1 G93A mice. SOD1 G93A... | Download Scientific Diagram